➣➣This article will tell you about subacute combined degeneration and the knowledge points corresponding to the clinical manifestations of subacute combined degeneration. I hope it will be helpful to you , don't forget to bookmark this site. Article catalog: 1. 2012 Clinical Practitioner Examination Guidance: Subacute Combined Spinal Cord Degeneration 2. Can Subacute Combined Spinal Cord Degeneration Be Cured? 3. Subacute Combined Spinal Cord Degeneration 4. Differential Diagnosis of Subacute Combined Spinal Cord Degeneration 2012 Clinical Practitioner Examination Counseling: Subacute combined degeneration of the spinal cord Subacute combined degeneration of the spinal cord is a degenerative disease of the nervous system caused by vitamin B12 deficiency. Mainly, peripheral sensory disturbance often accompanied by peripheral nerve damage. Subacute combined degeneration of the spinal cord (SCD), referred to as subacute combined degeneration (SCD), is a disease of central and peripheral nervous system degeneration caused by insufficient content in the body due to vitamin B12 intake, absorption, binding, transport or metabolic disorders. Damage to the posterior cord of the spinal cord gradually leads to clumsy limb movements, easy falls, a feeling of stepping on cotton when walking, and difficulty in closing eyes or walking in the dark. The physical examination showed that the sensation of tuning fork vibration and joint position of the lower limbs decreased or disappeared, walking unsteadily, gait faltering, widening of gait base, positive Romberg sign, etc. Can subacute combined degeneration of the spinal cord be cured? Subacute combined spinal cord degeneration can be cured. Subacute combined degeneration of the spinal cord is a neurodegenerative disease. The cause of this disease is associated with neurotrophic factors caused by vitamin deficiency. The key to treating this disease lies in early control. Analysis: According to your description, it can be considered as subacute combined degeneration of the spinal cord. Subacute combined degeneration is treatable, and I've treated a few cases recently with good results. But whether your mother's subacute combined degeneration of the spinal cord is not sure from the information you provided. More detailed information is suggested. Early diagnosis and timely treatment are the key to improving the prognosis of this disease. If active treatment can be performed within 3 months of onset, most patients can recover completely; if adequate treatment persists for 6 months to 1 year, neurological dysfunction is difficult to recover. Without active treatment, neurological symptoms will continue to worsen, and even death may occur. Vitamin B12 is an essential vitamin in the synthesis of myelin sheath of human nerve cells. Its deficiency will lead to dysfunction of the synthesis of human nerve myelin sheath, which will lead to subacute combined degeneration. Subacute combined degeneration of the spinal cord can be considered to strengthen the functional exercise of paralyzed limbs, supplemented by acupuncture and physical therapy. Subacute combined degeneration Subacute combined degeneration is also called vitamin deficiency syndrome, and there are many causes of cervical spondylosis. MRI can completely diagnose it. Subacute combined degeneration (subacute bined degeneration), also known as vitamin B12 neuropathy, is caused by vitamin B12 deficiency. It is more common in adults over the age of 40. Both men and women can be affected. Slowly progressive degeneration mainly occurs in the dorsal cord, lateral cord and peripheral nerves of the spinal cord. Subacute combined degeneration of the spinal cord is mainly caused by vitamin B12 deficiency, which is related to too little vitamin intake or absorption and metabolism disorders. Most of them are seen after gastrectomy or atrophic gastritis. The main treatment is vitamin B12 supplementation. Subacute combined degeneration of the spinal cord is a neurodegenerative disease caused by vitamin B12 deficiency. The lesion mainly involves the dorsal cord, lateral cord, and peripheral nerves. The clinical manifestations are deep sensory loss of both lower extremities, sensory ataxia, spastic paraplegia, and peripheral neuropathy. Most of the onset is above middle age, and there is no significant difference between men and women. The differential diagnosis of subacute combined degeneration of the spinal cord is based on the slow and insidious onset of subacute combined degeneration after middle age, subacute or chronic disease course, combined damage to the dorsal cord, pyramidal tract and peripheral nerves, decreased serum vitamin B12, and vitamin B12 treatment The diagnosis can be made after improvement of neurological symptoms. Vitamin B12 deficiency combined with spinal cord dysfunction can lead to subacute combined spinal cord degeneration. Cerebrospinal fluid examination by lumbar puncture was normal. Spinal cord MRI can reveal abnormal signals of subacute combined degeneration of the spinal cord in degenerated segments. Diagnosis and Differential Diagnosis According to the onset after middle age, symptoms and signs of spinal cord, pyramidal tract and peripheral nerve damage, combined with anemia, and neurological symptoms improved after vitamin B12 treatment, the diagnosis can be made. Peripheral blood and bone marrow smear examination (1) For patients with anemia, peripheral blood and bone marrow smears suggest giant cell hypochromic anemia, decreased blood reticulocyte count, and increased jaundice index. Nystagmus, cerebellar sign, pyramidal tract sign, posterior chordal dysfunction, performance of asymmetric peripheral nerve damage, cerebrospinal fluid examination, evoked potential, head CT and MRI are helpful for diagnosis. That’s all for the introduction of subacute combined degeneration. Thank you for taking the time to read the content of this site. Don’t forget to search for more information about the clinical manifestations of subacute combined degeneration and subacute combined degeneration.

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